skip to primary navigationskip to content

Cambridge Institute for Medical Research

Department A-Z

Our strategy

We aim to create an inspiring environment in which outstanding scientists can excel. By providing state-of-the-art core facilities and support for our researchers, we foster new collaborations that spark discoveries about fundamental cellular processes and their relevance in disease.

Read more

Research advances

Reporting in Nature Genetics, the Lehner lab have revealed that MORC2 is essential for epigenetic silencing by the HUSH complex, which is altered in Charcot-Marie-Tooth disease.

Read more

Research advances

In Science, Fanni Gergely (CRUK Cambridge Institute) has collaborated with Geoff Woods (CIMR), Ian Goodfellow (Dept Pathology) and others to reveal how Zika virus might target the developing embryonic brain, leading to microcephaly.

Read more

Distinguished Career Award

Many congratulations to Jim Huntington (right) who has received a 2017 BACH Distinguished Career Award from The International Society on Thrombosis and Haemostasis (ISTH).

Read more

Spotlight on Future Therapeutics

Gillian Griffiths and colleagues discuss new insights being gained into the awakening of our immune system self-defence force.

Read more

New Royal Society Fellows

Many congratulations to David Rubinsztein and David Owen for their election to the Royal Society.

Read more


A unique partnership between basic and clinical research, aiming to understand the cellular basis of disease






New publications

 

Weekes lab (Cell Rep. 2017)

Temporal proteomics of EBV replication.

 

Read and Deane labs (Nature Comm. 2017)

Structural insights into Hunter syndrome.

 

Lehner lab (Nature Genetics 2017)

MORC2 in epigenetic silencing by HUSH complex.

 

Buss lab (Cell Rep. 2017)

MYO6 organization of signalling endosomes.

 

Ron and Marciniak labs (eLife 2017)

Reassessing PPP1R15A regulation.

 

Rubinsztein lab (Nature 2017)

PolyQ protein control of autophagy.

 

Reid lab (J. Cell Biol. 2017)

ER-endosome contacts and lysosome function in HSPs.