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Cambridge Institute for Medical Research

Department A-Z

Our strategy

We aim to create an inspiring environment in which outstanding scientists can excel. By providing state-of-the-art core facilities and support for our researchers, we foster new collaborations that spark discoveries about fundamental cellular processes and their relevance in disease.

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Research advances

In FASEB Journal, the Marciniak lab reveal that accumulation of mutant α1-antitrypsin polymers, implicated in liver disease, leads to fragmentation of the ER, but does not prevent SNARE-mediated vesicular transport.

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Research advances

The Robinson lab report in eLife that the antiviral protein tetherin also attaches secreted exosomes to the cell surface, providing a potential means of control over exosome-mediated communication.

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New approaches to type 1 diabetes

Clinical research led by Frank Waldron-Lynch suggests that limiting autoimmune damage against insulin-producing cells may be an effective approach to treating type 1 diabetes.

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Dementia: catching the memory thief

A new video highlighting Cambridge research into dementia, including David Rubinsztein and Peter St George-Hyslop.

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FEBS National Lecture Award

Congratulations to Alan Warren for receipt of the FEBS National Lecture Award 2016, presented by Angela Sessitsch, President of the Austrian Society for Genetics, Molecular Biology and Biotechnology.

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A unique partnership between basic and clinical research, aiming to understand the cellular basis of disease

New publications


Lehner lab (Cell Rep. 2016)

ATF7IP is a key effector of the HUSH complex.


Robinson lab (eLife 2016)

Tetherin as an exosomal tether.


Marciniak lab (FASEB J. 2016)

ER transport after fragmentation.


Luzio lab (Curr. Biol. 2016)

Re-evaluating the lysosome regeneration cycle.


Lehner lab (Traffic 2016)

Trafficking control of tetherin.


Rubinsztein lab (Nature Comm. 2016)

Autophagy-lysosome dysfunction in Parkinson's disease.